Stephan Guyenet: a list of contradictions/inconsistencies, deception/lies.
I'm sort of tired with the nonchalant way he spouts contradictions, inconsistencies, deceptive statements and overt lies (which he never clarifies or addresses).
This is a problem because many impressionable sick overweight diabetic individuals look up to him for guidance, meanwhile he is a lying / being deceptive to preserve his theory. That's pretty horrid. He pimps himself out as a VIP authority on the cutting edge of obesity research; naturally he has garnered a devout following of fat diabetic people looking for answers. In reality he is borderline mercola-ish in his kooky illogical beliefs; he gets away with it because he cleans up well, sounds like he makes sense when he spouts off, and has a PhD.
I have decided to maintain a blog post which will serve the purpose of CAPTURING AND ISOLATING the numerous contradictions and inconsistencies and deceptive statements uttered by Stephan Guyenet. That will be this post.
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Inconsistency/contradiction: Belief in set point theory co-existing with acknowledging sustained body fat growth from hypoglycemia
Deception: Using the case of Jenny Ruhl to support his belief of insulin as an anti-obesity hormone
Deception: Using a 30 year old study of low dose insulin in rats as evidence that insulin prevents obesity. This is deceptive because he is not mentioning the vast majority of evidence in humans supports the opposite conclusion. He is misleading his readers into believing that this 30 year old study is typical when investigating the relationship between insulin and body fat. No medical practitioner, diabetes expert, or diabetic patient will affirm Guyenet's crazy idea that insulin therapy inhibits body fat. It is generally well accepted that insulin therapy leads to body fat gain even outside of frequent hypoglycemic episodes.
Deception: Using the case of type II diabetic weight loss with amylin administered with short acting insulin as evidence that insulin is not intrinsically fattening.
(this will be updated frequently, stay tuned!)
5 comments:
- Stipetic said...
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These two posts line up the dominos pretty well. You really have an uncanny grasp of reality.
- ItsTheWooo said...
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Thxu!
I feel like this blog post will be very necessary. Too often Guyenet gets away with spouting off, and then just casually modifying his views / contradicting himself. You can't call yourself a VIP of obesity research PhD rockstar and pull that crap. You have many, many people listening to you and looking up to you. This blog entry is very necessary. - gallier2 said...
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You can add following comment in your list
http://wholehealthsource.blogspot.com/2012/01/insulin-and-obesity-another-nail-in.html?showComment=1328154692262#c7816253102359327910
which is extremely clear how far off he's gone. 38% percent body fat is huge difference especially if the lean mass atrophied accordingly.
This capacity to wipe such inconvenient facts under the rug with a straight face would be funny if it wasn't so sad.
Reminds me of CarbSane some months ago, when she asserted that DNL is negligible in obesity. She then put up a blog entry to "refute" my counter that it isn't negligible especially in presence of fructose.
She put up some link to studies of which one was really interesting. It showed without a shadow of a doubt that in young healthy individuals on a quite balanced diet (not a couch potatoe diet, it had around 160g of carbs a day, which is even bordering low-carb) there was at least 10% of the trigs entering fat cells that came from DNL. The 10% was a mean over all people on the diet, the highest guy had around 30%. The method used had an asymptotic behaviour, this mean that had the study continued the proportion would have still gone up. The reasearcher thought that they cut off the study a bit too early but that they weren't far from the baseline. It's obvious from that study that if you take an obese patient on the SAD diet you will land on much, much more DNL. Nothing negligible at all.
